ZHAO Yimeng
Institute of Cardiovascular Disease, University of South China & Key Laboratory for Arteriosclerology of Hunan Province & Human International Scientific and Technological Cooperation Base of Arteriosclerotic Disease, Hengyang, Hunan 421001, ChinaBAI Xueqin
Institute of Cardiovascular Disease, University of South China & Key Laboratory for Arteriosclerology of Hunan Province & Human International Scientific and Technological Cooperation Base of Arteriosclerotic Disease, Hengyang, Hunan 421001, ChinaHE Junfeng
Institute of Cardiovascular Disease, University of South China & Key Laboratory for Arteriosclerology of Hunan Province & Human International Scientific and Technological Cooperation Base of Arteriosclerotic Disease, Hengyang, Hunan 421001, ChinaWANG Ping
Changde Wuling District Center for Disease Control and Prevention, Changde, Hunan 415000, ChinaLIU Lushan
Institute of Cardiovascular Disease, University of South China & Key Laboratory for Arteriosclerology of Hunan Province & Human International Scientific and Technological Cooperation Base of Arteriosclerotic Disease, Hengyang, Hunan 421001, China1.Institute of Cardiovascular Disease, University of South China & Key Laboratory for Arteriosclerology of Hunan Province & Human International Scientific and Technological Cooperation Base of Arteriosclerotic Disease, Hengyang, Hunan 421001, China;2.Changde Wuling District Center for Disease Control and Prevention, Changde, Hunan 415000, China)
R363;R5
ZHAO Yimeng, BAI Xueqin, HE Junfeng, WANG Ping, LIU Lushan. Effect of ApoER2 degradation by PCSK9 on the anti-inflammatory effect of ApoE/ApoER2[J]. Editorial Office of Chinese Journal of Arteriosclerosis,2023,31(5):375-382.
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