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大气细颗粒物对动脉粥样硬化影响的研究进展
作者:
作者单位:

(1.中国环境科学研究院 环境基准与风险评估国家重点实验室, 北京市 100012;2.包头医学院公共卫生学院, 内蒙古包头市 014040)

作者简介:

王菲菲,博士,副研究员,研究方向为环境污染物对人体健康的损伤效应及机制研究,E-mail为wangff@craes.org.cn。


Research Progress on the Effect of Air Fine Particulate Matter on Atherosclerosis
Author:
Affiliation:

1.Chinese Research Academy of Environmental Sciences & State Key Laboratory of Environmental Criteria and Risk Assessment, Beijing 100012, China;2.School of Public Health, Baotou Medical College, Baotou, Inner Mongolia 014040, China)

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    摘要:

    介绍了近年来国内外大气细颗粒物(PM2.5)促进动脉粥样硬化发生发展的流行病学和毒理学研究进展。通过对关键指标的提炼汇总,为相关领域研究者全面掌握该领域现阶段研究全貌和未来研究趋势,并为选择下一步研究方向提供参考和依据。

    Abstract:

    This review summarized the recent research progress on the effect of air fine particulate matter on atherosclerosis, including human epidemiological and experimental laboratory studies. Through extracting and analysing key indicators, the review can provide a perspective and comprehensive background reference for further research in the field.

    参考文献
    [1] Loftus C, Yost M, Sampson P, et al.Regional PM2.5 and asthma morbidity in an agricultural community:A panel study.Environ Res, 5,6:505-512.
    [2] Raaschou-Nielsen O, Andersen ZJ, Beelen R, et al.Air pollution and lung cancer incidence in 17 European cohorts:prospective analyses from the European Study of Cohorts for Air Pollution Effects (ESCAPE).Lancet Oncol, 3,4 (9):813-822.
    [3] Brook RD, Franklin B, Cascio W, et al.Air pollution and cardiovascular disease:a statement for healthcare professionals from the Expert Panel on Population and Prevention Science of the American Heart Association.Circulation, 4,9 (21):2 655-671.
    [4] Yatera K, Hsieh J, Hogg JC, et al.Particulate matter air pollution exposure promotes recruitment of monocytes into atherosclerotic plaques.Am J Physiol Heart Circ Physiol, 8,4 (2):H 944-953.
    [5] Sun Q, Hong X, Wold LE.Cardiovascular effects of ambient particulate air pollution exposure.Circulation, 0,1 (25):2 755-765.
    [6] Araujo JA, Nel AE.Particulate matter and atherosclerosis:role of particle size, composition and oxidative stress.Part Fibre Toxicol, 9,6 (1):24-42.
    [7] Künzli N, Jerrett M, Mack WJ, et al.Ambient air pollution and atherosclerosis in Los Angeles.Environ Health Perspect, 5,3 (2):201-206.
    [8] Mack WJ, LaBree L, Liu CR, et al.Correlations between measures of atherosclerosis change using carotid ultrasonography and coronary angiography.Atherosclerosis, 0,0 (2):371-379.
    [9] Howard G, Wagenknecht LE, Burke GL, et al.Cigarette smoking and progression of atherosclerosis:The Atherosclerosis Risk in Communities (ARIC) Study.JAMA, 8,9 (2):119-124.
    [10] Perez L, Wolf K, Hennig F, et al.Air pollution and atherosclerosis:a cross-sectional analysis of four European cohort studies in the ESCAPE study.Environ Health Perspect, 5,3 (6):597-605.
    [11] Su TC, Hwang JJ, Shen YC, et al.Carotid intima-media thickness and long-term exposure to traffic-related air pollution in middle-aged residents of Taiwan:a cross-sectional study.Environ Health Perspect, 5,3 (8):773-778.
    [12] Roux AVD, Auchincloss AH, Franklin TG, et al.Long-term exposure to ambient particulate matter and prevalence of subclinical atherosclerosis in the Multi-Ethnic Study of Atherosclerosis.Am J Epidemiol, 8,7(6):667-675.
    [13] Adar SD, Sheppard L, Vedal S, et al.Fine particulate air pollution and the progression of carotid intima-medial thickness:a prospective cohort study from the multi-ethnic study of atherosclerosis and air pollution.PLoS Med, 3,0 (4):e1001430.
    [14] Hoffmann B, Moebus S, Krger K, et al.Residential exposure to urban air pollution, ankle-brachial index, and peripheral arterial disease.Epidemiology, 9,0 (2):280-288.
    [15] Akintoye E, Shi L, Obaitan I, et al.Association between fine articulate matter exposure and subclinical atherosclerosis:A meta-analysis .Eur J Prev Cardiol, 5,3 (6):602-612.
    [16] Provost EB, Madhloum N, Panis LI, et al.Carotid intima-media thickness, a marker of subclinical atherosclerosis, and particulate air pollution exposure:the meta-analytical evidence.PLoS One, 5,0 (5):e0127014.
    [17] Sun M, Kaufman JD, Kim SY, et al.Particulate matter components and subclinical atherosclerosis:common approaches to estimating exposure in a Multi-Ethnic Study of Atherosclerosis cross-sectional study.Environ Health, 3,2:39-41.
    [18] Kim SY, Sheppard L, Kaufman JD, et al.Individual-level concentrations of fine particulate matter chemical components and subclinical atherosclerosis:a cross-sectional analysis based on 2 advanced exposure prediction models in the multi-ethnic study of atherosclerosis.Am J Epidemiol, 4,0 (7):718-728.
    [19] Allen RW, Criqui MH, Roux AVD, et al.Fine particulate matter air pollution, proximity to traffic, and aortic atherosclerosis.Epidemiology, 9,0 (2):254-264.
    [20] Hoffmann B, Moebus S, Mhlenkamp S, et al.Residential exposure to traffic is associated with coronary atherosclerosis.Circulation, 7,6(5):489-496.
    [21] Künzli N, Jerrett M, Garcia-Esteban R, et al.Ambient air pollution and the progression of atherosclerosis in adults.PloS One, 0,5 (2):e9096.
    [22] Klsch H, Hennig F, Moebus S, et al.Are air pollution and traffic noise independently associated with atherosclerosis:the Heinz Nixdorf Recall Study.Eur Heart J, 4,5 (13):853-860.
    [23] Painschab MS, Davila-Roman VG, Gilman RH, et al.Chronic exposure to biomass fuel is associated with increased carotid artery intima-media thickness and a higher prevalence of atherosclerotic plaque.Heart, 3,9 (14):984-991.
    [24] Fuks K, Moebus S, Hertel S, et al.Long-term urban particulate air pollution, traffic noise, and arterial blood pressure.Environ Health Perspect, 1,9 (12):1 706-711.
    [25] Fan J, Qin X, Xue X, et al.Effects of carbon components of fine particulate matter (PM2.5) on atherogenic index of plasma.Zhonghua Yu Fang Yi Xue Za Zhi, 4,8 (1):33-37.
    [26] Wu S, Yang D, Wei H, et al.Association of chemical constituents and pollution sources of ambient fine particulate air pollution and biomarkers of oxidative stress associated with atherosclerosis:A panel study among young adults in Beijing, China.Chemosphere, 5,5:347-353.
    [27] Wilker EH, Alexeeff SE, Suh H, et al.Ambient pollutants, polymorphisms associated with microRNA processing and adhesion molecules:the Normative Aging Study.Environ Health, 1,0:45-50.
    [28] Reddick R L, Zhang S H, Maeda N.Atherosclerosis in mice lacking ApoE.Evaluation of lesional development and progression.Arterioscler Thromb, 4,4 (1):141-147.
    [29] Sun Q, Wang A, Jin X, et al.Long-term air pollution exposure and acceleration of atherosclerosis and vascular inflammation in an animal model.JAMA, 5,4 (23):3 003-010.
    [30] Chen LC, Nadziejko C.Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice:V.CAPs exacerbate aortic plaque development in hyperlipidemic mice.Inhal Toxicol, 5,7(4-5):217-224.
    [31] 彭毅, 邵紫韫, 丁世芳.动脉粥样硬化中的细胞因子及细胞因子相关信号通路.中国动脉硬化杂志, 8,6 (2):161-164.
    [32] 陈临溪, 秦旭平, 黄秋临.血管内皮细胞药理与临床.北京:人民军医出版社, 2012; 64-69.
    [33] Wei H, Wei D, Yi S, et al.Oxidative stress induced by urban fine particles in cultured EA.hy926 cells.Hum Exp Toxicol, 1,0 (7):579-590.
    [34] Chen T, Jia G, Wei Y, et al.Beijing ambient particle exposure accelerates atherosclerosis in ApoE knockout mice.Toxicol Lett, 3,3 (2):146-153.
    [35] Rao X, Zhong J, Maiseyeu A, et al.CD36-dependent 7-ketocholesterol accumulation in macrophages mediates progression of atherosclerosis in response to chronic air pollution exposure.Circ Res, 4,5 (9):770-780.
    [36] Liu SW, Qiao SB, Yuan JS, et al.Association of plasma visfatin levels with inflammation, atherosclerosis and acute coronary syndromes (ACS) in humans.Clin Endocrinol (Oxf), 9,1 (2):202-207.
    [37] Yan JJ, Tang NP, Tang JJ, et al.Genetic variant in visfatin gene promoter is associated with decreased risk of coronary artery disease in a Chinese population.Clin Chim Acta, 0,1 (1):26-30.
    [38] Romacho T, Azcutia V, Vazquez-Bella M, et al.Extracellular PBEF/NAMPT/visfatin activates pro-inflammatory signalling in human vascular smooth muscle cells through nicotinamide phosphoribosyltransferase activity.Diabetologia, 9,2 (11):2 455-463.
    [39] Wan Q, Cui X, Shao J, et al.Beijing ambient particle exposure accelerates atherosclerosis in ApoE knockout mice by upregulating visfatin expression.Cell Stress Chaperones, 4,9 (5):715-724.
    [40] Sancini G, Farina F, Battaglia C, et al.Health risk assessment for air pollutants:alterations in lung and cardiac gene expression in mice exposed to milano winter fine particulate matter (PM2.5).PLoS One, 4,9 (10):e109685.
    [41] 徐贞贞, 张志红, 马晓燕, 等.PM2.5通过氧化应激对人支气管上皮细胞JAK/STAT信号通路的影响.卫生研究, 5,4 (3):451-455.
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引用本文

王菲菲,赵永东,聂静,钱岩,赵秀阁.大气细颗粒物对动脉粥样硬化影响的研究进展[J].中国动脉硬化杂志,2016,24(8):850~854.

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  • 收稿日期:2015-09-17
  • 最后修改日期:2016-02-06
  • 在线发布日期: 2016-07-25

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